high aluminum

[bmom]

My sons hair analysis showed high aluminum. He rarely drinks soda, so I am looking into different pots and pans. I am debating between anodized aluminum- which says does not enter food, stainless steel, etc. Any suggestions on what will help/not harm? Thanks again.

[patty]

We use stainless steel and cast iron. Glass like corning ware is also good. We rarely use nonstick unless we are making waffles. We also avoid nonstick & aluminum bakeware.

 

Patty

[Mustang Carole]

Hello,

DEODERANT has aluminum. , so check your brand and toss them.

 

Buy the non aluminum kind, like Jason's , etc. and can find them at Wholefoods

and Better Health stores or on line. They have some great fragrances that are

seasonal, and also the non fragrant, most are stick and a few roll on for male

and females. I love them and tossed the Arrid.

 

Mustang Carole

[kim]

This site might be helpful

 

http://www.bellaonline.com/articles/art7739.asp

 

don't miss clicking on 2nd page

 

Dangers of Aluminum Toxicity Part II

 

These are just a few of the alum studies that I have saved.

 

Some of these were copied because I was looking at the relationship to vaccines/ soy and my youngest sons apparent adversion to many protein containing foods. They may not be of interest to all.

 

http://www.ingentaconnect.com/content/hum/...000002/art00007

Effect of Aluminum on the Blood-Brain Barrier Permeability in Acute and Chronically Hyperglycemic Rats

 

 

 

http://findarticles.com/p/articles/mi_m322...71/ai_n13684250

http://www.urmc.rochester.edu/pr/news/story.cfm?id=981

http://www.medicalnewstoday.com/medicalnews.php?newsid=35136

http://www.aafp.org/afp/20050315/tips/11.html

http://www.sciencedaily.com/releases/2005/...51218111320.htm

 

 

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...p;dopt=Abstract

 

Aluminum transfer as glutamate complex through blood-brain barrier. Possible implication in dialysis encephalopathy.

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...st_uids=7779551

 

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...t_uids=12657166

 

Dietary protein restriction causes modification in aluminum-induced alteration in glutamate and GABA system of rat brain.Nayak P, Chatterjee AK.

Department of Physiology, Sikkim Manipal Institute of Medical Sciences, 5th Mile, Tadong, Gangtok 737102, Sikkim, India. nprasunpriya@hotmail.com

 

BACKGROUND: Alteration of glutamate and gamma-aminobutyrate system have been reported to be associated with neurodegenerative disorders and have been postulated to be involved in aluminum-induced neurotoxicity as well. Aluminum, an well known and commonly exposed neurotoxin, was found to alter glutamate and gamma-aminobutyrate levels as well as activities of associated enzymes with regional specificity. Protein malnutrition also reported to alter glutamate level and some of its metabolic enzymes. Thus the region-wise study of levels of brain glutamate and gamma-aminobutyrate system in protein adequacy and inadequacy may be worthwhile to understand the mechanism of aluminum-induced neurotoxicity. RESULTS: Protein restriction does not have any significant impact on regional aluminum and gamma-aminobutyrate contents of rat brain. Significant interaction of dietary protein restriction and aluminum intoxication to alter regional brain glutamate level was observed in the tested brain regions except cerebellum. Alteration in glutamate alpha-decarboxylase and gamma-aminobutyrate transaminase activities were found to be significantly influenced by interaction of aluminum intoxication and dietary protein restriction in all the tested brain regions. In case of regional brain succinic semialdehyde content, this interaction was significant only in cerebrum and thalamic area. CONCLUSION: The alterations of regional brain glutamate and gamma-aminobutyrate levels by aluminum are region specific as well as dependent on dietary protein intake. The impact of aluminum exposure on the metabolism of these amino acid neurotransmitters are also influenced by dietary protein level. Thus, modification of dietary protein level or manipulation of the brain amino acid homeostasis by any other means may be an useful tool to find out a path to restrict amino acid neurotransmitter alterations in aluminum-associated neurodisorders.

 

PMID: 12657166 [PubMed - indexed for MEDLINE]

 

 

 

 

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...t_uids=12197946

 

Response of regional brain glutamate transaminases of rat to aluminum in protein malnutrition.Nayak P, Chatterjee AK.

Department of Physiology, Sikkim Manipal Institute of Medical Sciences, 5th Mile, Tadong, Gangtok 737 102, Sikkim, India. nprasunpriya@hotmail.com

 

BACKGROUND: The mechanism of aluminum-induced neurotoxicity is not clear. The involvement of glutamate in the aluminium-induced neurocomplications has been suggested. Brain glutamate levels also found to be altered in protein malnutrition. Alterations in glutamate levels as well as glutamate-alpha-decarboxylase in different regions of rat brain has been reported in response to aluminum exposure. Thus the study of glutamate metabolising enzymes in different brain regions of rats maintained on either normal or restricted protein diet may be of importance for understanding the neurotoxicity properties of aluminium. RESULTS: Dietary protein restrictions does not have an significant impact on regional aluminum content of the brain. The interaction of aluminum intoxication and protein restriction is significant in the thalamic area and the midbrain-hippocampal region in cases of glutamate oxaloacetate transaminase. In the case of glutamate pyruvate transaminase, this interaction is significant only in thalamic area. CONCLUSION: The metabolism of amino acids, as indicated by activities of specific transaminases, of brain is altered in response to aluminum exposure. These alterations are region specific and are dependent on dietary protein intake or manipulation of the brain amino acid homeostasis.

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...t_uids=11696403

 

Effects of aluminium exposure on brain glutamate and GABA systems: an experimental study in rats.Nayak P, Chatterjee AK.

Biochemistry and Nutrition Research Laboratory, Department of Physiology, University of Calcutta, 92 A.P.C. Road, 700 009, Calcutta, India. nprasunpriya@hotmail.com

 

It has been postulated that the neurotoxic effects of aluminium could be mediated through glutamate, an excitatory amino acid. Hence the effects of aluminium administration (at a dose of 4.2mg/kg body weight daily as aluminium chloride, hexahydrate, intraperitoneally, for 4 weeks) on glutamate and gamma-amino butyrate (GABA), an inhibitory amino acid, and related enzyme activities in different regions of the brain were studied in albino rats. The glutamate level increased significantly in the cerebrum, thalamic area, midbrain-hippocampal region and cerebellum in response to in vivo aluminium exposure. The aluminium insult also caused significant increases in glutamate alpha-decarboxylase activity in all the brain regions. However, on aluminium insult, the GABA content was not significantly changed except in the thalamic area, where it was elevated. On the contrary, the GABA-T activities of all the regions were reduced significantly in all regions except the midbrain-hippocampal region. However, the succinic semi-aldehyde content of all brain regions increased, often significantly. The aluminium-induced modification of the enzyme activities may be either due to the direct impact of aluminium or due to aluminium-induced changes in the cellular environment. The aluminium-induced differential regional accumulation of glutamate or other alterations in enzymes of the glutamate-GABA system may be one of the causes of aluminium-induced neurotoxicity.

 

 

http://www.ncbi.nlm.nih.gov/entrez/query.f...t_uids=11346489

 

Differential responses of certain brain phosphoesterases to aluminium in dietary protein adequacy and inadequacy.Nayak P, Chatterjee AK.

Biochemistry and Nutrition Research Laboratory, Department of Physiology, University of Calcutta, 92 A.P.C. Road, Calcutta 700 009, India. nprasunpriya@hotmail.com

 

The aluminium-induced neurotoxic consequences include, among other factors, dephosphorylation, phosphorylation as well as hyperphosphorylation of specific macromolecules. Accordingly, activities of phosphoesterases were measured in different regions of rat brain, maintained with either adequate or inadequate protein diet, following aluminium exposure. Male Wistar rats weighing 80-100 g were treated with aluminium chloride at a dose of 15% of the LD50 for 4 weeks. In different regions of the brain of aluminium-exposed rats, significant variation in both phosphomonoesterase and phosphodiesterase activities have been recorded. These alterations were found to be varied when the rats were subjected to dietary protein insufficiency. These findings demonstrate the specificity of aluminium on different phosphoesterases. These regional variations may be attributed to the accumulated level of aluminium or may be due to cellular localization of these enzymes and linked to whether the enzymes are compartmentalized with different aluminium hydration species.