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by Aubrey K. Ewing, Ph.D.
One of the most confounding conditions encountered by pediatricians,
neurologists, mental health professionals, and educators is Attention
Deficit Disorder, with or without Hyperactivity (ADD/ADHD). Once simply
referred to as hyperactivity or minimal brain dysfunction, we now know
that this condition is far more complex than those names imply. While
there has been considerable speculation in the scientific literature as
to the etiology of ADD/ADHD, only recently have we begun to understand
that those suffering from attention disorders are neurologically different
from the normal population. Two particularly compelling studies elucidate
the neurobiological difference between those with attention disorders
and normal populations.
Related Symptoms That Can Be Treated
with Neurofeedback |
Impulsiveness
Lack of attention, focus,
and concentration
High activity level
Poor task completion and organizational skills
|
Reported Results of Treatment |
Higher intelligence
test scores
Greater realization
of innate potential
Improved behavior
and learning
Improved school grades
Better job performance
Increased self-esteem
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Reduced Glucose Metabolism
The first was published in the New
England Journal of Medicine in November 1990. It reported the
results of a positron emission tomography (PET) scan study of adults who
had been diagnosed with ADHD as children (Zametkin, et al.). The study
showed that glucose metabolism in the brains of these adults was significantly
reduced, particularly in areas of the brain associated with control of
attention and motor activity.
Theta > Beta Wave Activity
A second study, published in Pediatric Neurology
in February 1992, distinguished ADHD boys from a control group, using
a topographic (EEG) brain-mapping protocol (Mann, et al.). The study indicated
that ADHD subjects consistently produced more theta (4 7.75hz)
slow EEG activity, and less beta I (12.75 21hz) fast-wave activity.
Theta is associated with inattention, distractibility, disorganization,
and poor task sequencing. Beta correlates with the ability to focus, organize,
pay attention, sequence, and associate cause and effect.
ADHD clearly has a strong biologic substrate, and cortical activation
is apparently decreased in ADHD. It is with this understanding, and the
development of sophisticated computer hardware and software, that a neurobehavioral
treatment for ADHD using EEG technology has been developed.
Requirements of Training
Neurobehavioral treatment of ADHD essentially requires
patients to learn to normalize their EEGs. Patients train themselves to
increase production of beta I while inhibiting theta and electromyographic
activity (muscle activity or movement). This treatment protocol, developed
by Dr. Joel Lubar, a psychophysiologist at the University of Tennessee,
has been successful as an adjunct to traditional approaches to managing
ADHD. In outcome studies that have been replicated in many clinical settings
around the country, patients typically increase IQ scores by 10 to 15
points and improve significantly in measures of attentiveness, hyperactivity,
and academic performance. Further, these positive EEG changes appear to
be long-term.
This treatment of ADHD requires a significant commitment of time and
resources by patients and their families. The treatment program generally
requires 35 to 50 work sessions with the EEG instrument and, in the case
of children, initial involvement by their parents. (Neurobehavioral treatment
is effective with adult ADHD as well.) Some insurance companies may not
cover costs, in which case a financial commitment is also necessary.
| The clinical results employing EEG biofeedback
training have been replicated by more than 200 organizations internationally. |
Some children are able to reduce and even eliminate the need for medication.
The clinical results employing EEG biofeedback training have been replicated
by more than 200 organizations internationally. Published clinical studies
exist, and controlled studies are being planned or carried out to determine
the effectiveness of EEG biofeedback training as compared to medication,
behavior therapy, and other techniques. Additional research is needed
to determine the extent of long-term benefits.
Many children with mild to moderate learning disabilities can respond
very well to EEG biofeedback, provided material that is appropriate for
their particular learning disability is integrated into the treatment program and directly used in conjunction with the biofeedback
training itself. Learningdisabled persons without an accompanying diagnosis
of ADD/ADHD are not particularly good candidates for biofeedback training.
The Power of EEG Training
Though EEG training can be very powerful, it has
been shown to be even more effective when integrated into a multiple-component
treatment program, which may include a certain amount of behavior management,
medication if required, and parent training in management techniques.
When specific criteria are used to screen candidates,
success is reported for 80% of clients. Success is measured and validated
by testing, independent observation, and reports by teachers, parents,
and other health care professionals.
Who is a Good Candidate
for Neurofeedback Therapy?
Anyone with a primary diagnosis of ADD or ADHD, between
the ages of 7 to 50, with at least average intelligence.
Who is Not a Good Candidate
for This Therapy?
Treatment generally should not be offered if the person
also has any of these conditions:
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Childhood psychosis
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Significant seizure disorder where medications interfere with learning
(i.e., sedating medications)
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Severe depressive or bipolar illness
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Highly dysfunctional families who refuse to participate in indicated
therapy
Aubrey K.
Ewing, Ph.D., is board certified in applied psychophysiology
and biofeedback by the Biofeedback Certification Institute of America. He
is a certified counseling psychologist and licensed mental health
counselor.
Aubrey K.
Ewing, Ph.D. & Associates, P.A.
Boynton Beach, FL 33435
website:
www.drewingonline.com
Zametkin AJ,
Nordahl TE, Gross M, et al.
“Cerebral Glucose Metabolism
in Adults with Hyperactivity of Childhood Onset.”
New England Journal of
Medicine, 1990;
323:1361-6
Mann CA, Lubar, JF,
Zimmerman, AW, et al. “Quantitative Analysis of EEG in Boys with
Attention Deficit Hyperactivity Disorder; Controlled study with Clinical
Implications.” Pediatric Neurology, 1992; 8, 1:30-6
 Reprinted from
Latitudes,
vol. 1, no. 5; published by ACN.
Copyright ©2007 Association for Comprehensive NeuroTherapy. All Rights
Reserved.
Latitudes is a quarterly online publication of the
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non-toxic approaches to autism, depression, obsessive compulsive disorder and
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